Authors

Qiang He, West China School of Medicine/West China Hospital of Sichuan University
Wenjing Wang, West China School of Medicine/West China Hospital of Sichuan University
Dingkang Xu, Chinese Academy of Medical Sciences & Peking Union Medical College
Yang Xiong, West China School of Medicine/West China Hospital of Sichuan University
Chuanyuan Tao, West China School of Medicine/West China Hospital of Sichuan University
Chao You, West China School of Medicine/West China Hospital of Sichuan University
Lu Ma, West China School of Medicine/West China Hospital of Sichuan University
Junpeng Ma, West China School of Medicine/West China Hospital of Sichuan University
Caroline M. Nievergelt, Department of Psychiatry
Adam X. Maihofer, Department of Psychiatry
Torsten Klengel, Harvard Medical School
Elizabeth G. Atkinson, Broad Institute
Chia Yen Chen, Broad Institute
Karmel W. Choi, Broad Institute
Jonathan R.I. Coleman, King's College London
Shareefa Dalvie, University of Cape Town
Laramie E. Duncan, Stanford University
Mark W. Logue, National Center for PTSD
Allison C. Provost, Cohen Veterans Bioscience
Andrew Ratanatharathorn, Harvard T.H. Chan School of Public Health
Murray B. Stein, Department of Psychiatry
Katy Torres, Department of Psychiatry
Allison E. Aiello, The University of North Carolina at Chapel Hill
Lynn M. Almli, Carter Consulting, Incorporated
Ananda B. Amstadter, Virginia Institute for Psychiatric and Behavioral Genetics
Søren B. Andersen, Danish Veteran Centre
Ole A. Andreassen, Universitetet i Oslo
Paul A. Arbisi, Minneapolis Veterans Affairs Health Care System
Ariane Rung, LSU Health Sciences Center - New OrleansFollow
et al

Document Type

Article

Publication Date

1-31-2024

Publication Title

Translational Psychiatry

Abstract

Background: The causal effects of gut microbiome and the development of posttraumatic stress disorder (PTSD) are still unknown. This study aimed to clarify their potential causal association using mendelian randomization (MR). Methods: The summary-level statistics for gut microbiome were retrieved from a genome-wide association study (GWAS) of the MiBioGen consortium. As to PTSD, the Freeze 2 datasets were originated from the Psychiatric Genomics Consortium Posttraumatic Stress Disorder Working Group (PGC-PTSD), and the replicated datasets were obtained from FinnGen consortium. Single nucleotide polymorphisms meeting MR assumptions were selected as instrumental variables. The inverse variance weighting (IVW) method was employed as the main approach, supplemented by sensitivity analyses to evaluate potential pleiotropy and heterogeneity and ensure the robustness of the MR results. We also performed reverse MR analyses to explore PTSD’s causal effects on the relative abundances of specific features of the gut microbiome. Results: In Freeze 2 datasets from PGC-PTSD, eight bacterial traits revealed a potential causal association between gut microbiome and PTSD (IVW, all P < 0.05). In addition, Genus.Dorea and genus.Sellimonas were replicated in FinnGen datasets, in which eight bacterial traits revealed a potential causal association between gut microbiome and the occurrence of PTSD. The heterogeneity and pleiotropy analyses further supported the robustness of the IVW findings, providing additional evidence for their reliability. Conclusion: Our study provides the potential causal impact of gut microbiomes on the development of PTSD, shedding new light on the understanding of the dysfunctional gut-brain axis in this disorder. Our findings present novel evidence and call for investigations to confirm the association between their links, as well as to illuminate the underlying mechanisms.

PubMed ID

38296956

Volume

14

Issue

1

Comments

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Creative Commons License

Creative Commons Attribution 4.0 International License
This work is licensed under a Creative Commons Attribution 4.0 International License.

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