Insulin Resistance in Women Correlates with Chromatin Histone Lysine Acetylation, Inflammatory Signaling, and Accelerated Aging

Authors

Christina M. Vidal, City of Hope Comprehensive Cancer Center, Duarte, CA
Jackelyn A. Alva-Ornelas, City of Hope Comprehensive Cancer Center, Duarte, CA
Nancy Zhuo Chen, City of Hope Duarte, Duarte, CA
Parijat Senapati, City of Hope Comprehensive Cancer Center, Duarte, CA
Jerneja Tomsic, City of Hope Comprehensive Cancer Center, Duarte, CA
Vanessa Myriam Robles, City of Hope Comprehensive Cancer Center, Duarte, CA
Cristal Resto, City of Hope Comprehensive Cancer Center, Duarte, CA
Nancy Sanchez, City of Hope Comprehensive Cancer Center, Duarte, CA
Angelica Sanchez, City of Hope Comprehensive Cancer Center, Duarte, CA
Terry Hyslop, Sidney Kimmel Cancer Center, Philadelphia, PA
Nour Emwas, City of Hope Comprehensive Cancer Center, Duarte, CA
Dana Aljaber, City of Hope Comprehensive Cancer Center, Duarte, CA
Nick Bachelder, City of Hope Duarte, Duarte, CA
Ernest Martinez, University of California at Riverside, Riverside, CA
David Ann, City of Hope Comprehensive Cancer Center, Duarte, CA
Veronica Jones, City of Hope Comprehensive Cancer Center, Duarte, CA
Robert A. Winn, Virginia Commonwealth University, Richmond, VA
Lucio Miele, LSU Health Sciences Center - New OrleansFollow
Augusto C. Ochoa, LSU Health Sciences Center - New OrleansFollow
Eric C. Dietze, City of Hope Comprehensive Cancer Center, Duarte, CA
Rama Natarajan, City of Hope Duarte, Duarte, CA
Dustin Schones, City of Hope Duarte, Duarte, CA
Victoria L. Seewaldt, City of Hope Comprehensive Cancer Center, Duarte, CA

Document Type

Article

Publication Date

8-1-2024

Publication Title

Cancers

Abstract

BACKGROUND: Epigenetic changes link medical, social, and environmental factors with cardiovascular and kidney disease and, more recently, with cancer. The mechanistic link between metabolic health and epigenetic changes is only starting to be investigated. In our in vitro and in vivo studies, we performed a broad analysis of the link between hyperinsulinemia and chromatin acetylation; our top "hit" was chromatin opening at H3K9ac. METHODS: Building on our published preclinical studies, here, we performed a detailed analysis of the link between insulin resistance, chromatin acetylation, and inflammation using an initial test set of 28 women and validation sets of 245, 22, and 53 women. RESULTS: ChIP-seq identified chromatin acetylation and opening at the genes coding for TNFα and IL6 in insulin-resistant women. Pathway analysis identified inflammatory response genes, NFκB/TNFα-signaling, reactome cytokine signaling, innate immunity, and senescence. Consistent with this finding, flow cytometry identified increased senescent circulating peripheral T-cells. DNA methylation analysis identified evidence of accelerated aging in insulin-resistant vs. metabolically healthy women. CONCLUSIONS: This study shows that insulin-resistant women have increased chromatin acetylation/opening, inflammation, and, perhaps, accelerated aging. Given the role that inflammation plays in cancer initiation and progression, these studies provide a potential mechanistic link between insulin resistance and cancer.

PubMed ID

39123463

Volume

16

Issue

15

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