Authors

Mulu Z. Tesfay, University of Arkansas for Medical Sciences, Little Rock, AR
Yuguo Zhang, University of Arkansas for Medical Sciences, Little Rock, AR
Khandoker U. Ferdous, University of Arkansas for Medical Sciences, Little Rock, AR
Mika A. Taylor, University of Arkansas for Medical Sciences, Little Rock, AR
Aleksandra Cios, University of Arkansas for Medical Sciences, Little Rock, AR
Randal S. Shelton, University of Arkansas for Medical Sciences, Little Rock, AR
Camila C. Simoes, University of Arkansas for Medical Sciences, Little Rock, AR
Chelsae R. Watters, Mayo Clinic, Rochester, MN
Oumar Barro, Mayo Clinic, Rochester, MN
Natalie M. Elliott, Mayo Clinic, Rochester, MN
Bahaa Mustafa, University of Arkansas for Medical Sciences, Little Rock, AR
Jean Christopher Chamcheu, University of Louisiana Monroe, Monroe, LA
Alicia L. Graham, University of Arkansas for Medical Sciences, Little Rock, AR
Charity L. Washam, The Winthrop P. Rockefeller Cancer Institute, UAMS, Little Rock, AR
Duah Alkam, The Winthrop P. Rockefeller Cancer Institute, UAMS, Little Rock, AR
Allen Gies, The Winthrop P. Rockefeller Cancer Institute, UAMS, Little Rock, AR
Stephanie D. Byrum, The Winthrop P. Rockefeller Cancer Institute, UAMS, Little Rock, AR
Emmanouil Giorgakis, University of Arkansas for Medical Sciences, Little Rock, AR
Steven R. Post, University of Arkansas for Medical Sciences, Little Rock, AR
Thomas Kelly, University of Arkansas for Medical Sciences, Little Rock, AR
Jun Ying, UAMS College of Public Health, Little Rock, AR
Omeed Moaven, LSU Health Sciences Center - New OrleansFollow
Chiswili Y. Chabu, University of Missouri, Columbia, MO
Martin E. Fernandez-Zapico, Mayo Clinic, Rochester, MN
Dan G. Duda, Massachusetts General Hospital, Boston, MA
Lewis R. Roberts, Mayo Clinic, Rochester, MN
Rang Govindarajan, University of Arkansas for Medical Sciences, Little Rock, AR
Mitesh J. Borad, Mayo Clinic, Rochester, MN
Martin J. Cannon, The Winthrop P. Rockefeller Cancer Institute, UAMS, Little Rock, AR
et al

Document Type

Article

Publication Date

11-26-2024

Publication Title

Molecular Therapy Oncology

Abstract

Members of the Vesiculovirus genus including Jurona virus (JURV) have emerged as promising immunotherapeutic agents, characterized by their tumor selectivity, fast kinetics, low seroprevalence, and minimal toxicity in humans. Here, we demonstrate that the administration of JURV leads to tumor regression in both hepatocellular carcinoma (HCC) xenograft and syngeneic models. Furthermore, our findings indicate that combining JURV and anti-PD-1 therapy reduced tumor burden and improved survival rates over JURV or anti-PD-1 alone in an orthotopic HCC model. Proteogenomic analysis of JURV-treated, murine HCC tumors demonstrates that the therapeutic effects of the combination of JURV and anti-PD-1 are predominantly driven by coordinated activation of immune effectors, which modulate the tumor microenvironment into a state conducive to anti-tumor activity. Our results establish JURV as a potent candidate for immunovirotherapy in HCC, capable of modulating immune response and synergizing with standard of care for HCC to prolong survival in preclinical models. Further, this research deepens our understanding of JURV's anti-tumoral mechanisms and highlights its potential as a novel approach to HCC treatment strategies.

Volume

32

Issue

4

Comments

See article for full author list.

Creative Commons License

Creative Commons Attribution 4.0 International License
This work is licensed under a Creative Commons Attribution 4.0 International License.

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